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CPT1A can also protect cells from apoptosis by clearing the cytoplasmic long-chain fatty acyl-CoA such as palmitoyl-CoA, and thus impede the production of “palmitate/palmitoyl-CoA/ceramide” involved in the apoptosis pathway activation 31

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No clear phenotype has been associated with CAV2 deficiency in humans, while Cav2 knockout mice demonstrate severe pulmonary dysfunction along with little or no change in caveolin-1 expression and caveolae formation [46]