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This concept explains not only the susceptibility of KGDHC to TFEC-induced inactivation in rat kidney and cells in culture but also the susceptibility of aconitase and BCKADHC to thioacylation/inactivation

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The breakthrough for cagrilintide was applying the fatty acid acylation technology that made semaglutide viable—attaching a fatty acid chain to a modified amylin analog backbone that prevents aggregation while enabling extended albumin binding